Loss of β1 accessory Na+ channel subunits causes failure of carbamazepine, but not of lacosamide, in blocking high-frequency firing via differential effects on persistent Na+ currents Posted on 15. June 2018 by Kristian Reichelt Post navigation RIM3γ and RIM4γ are key regulators of neuronal arborizationThe presynaptic active zone protein RIM1α controls epileptogenesis following status epilepticus